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Long-term schisandrin B treatment (1–30 mg/kg/d for 15 days) in cerebral ischemia/reperfusion (I/R) injury [27] and gentamicin-induced nephrotoxicity [26] models in rats revealed that schisandrin B enhanced cerebral mitochondrial antioxidant status, preserved mitochondrial structural integrity, reduced Ca 2+ load and cytochrome c release, and protected from mPTP opening [26,27]